Prevent Age Stealers… Use these ’12 Keys’ to increase quality of life and get your glow back

Aging doesn’t wait until we are 50 years old. It starts when we misuse our body. Here are 12 keys to increase quality of life and prevent Age Stealers.

1- Age Stealers can target the lymph system. Organs with an outside arm of the lymphatic system include the skin, gut, and lungs.(1) Lymph formed in the liver is a major contributor to total lymph flow with as much as 50% of total lymph being from hepatic origin.(2) The lymphatic system helps rid the body of  toxins and waste. It helps to ditch the alcohol and smoking habit that can impair your lymph, liver, and skin health. PLUS, exercise such as jumping on the trampoline is excellent for cleaning all three.

2- Premature aging can begin in children suffering from obesity. In fact, obesity can not only be linked to poor learning skills and Alzheimer’s it can damage DNA. It may be time to seek out a certified health coach. Getting to the bottom of obesity and poor skin health requires that the conversation move  towards other factors that add to the obesity epidemic such as: depression, medications, pesticides, endocrine disruptors, and more. According to a German Study, there is a relationship between obesity, psychological factors and effective interventions.(3)

3-  Petroleum byproducts can be found in moisturizers, sunscreens and shampoos. Reduce and prevent premature aging with organic skin care products.

4- Inflammation is caused by consuming foods that are foreign to humans. Advanced glycation end-products (AGEs) are found to be linked to premature aging.  Various foods will cause various degrees of inflammation, and the most inflammatory producing foods are those that are grown using chemicals, pesticides and hormones, those that are injected with antibiotics, laced with sugars, and coated or stored to increase shelf -life. Inflammation in the skin and body can be reduced by going sugar-free, gluten-free, processed-free and eating more raw foods.

5- Increase emotional intelligence/ make mediation a habit. Meditation can keep you young in mind, body and spirit. When was the last time you turned off the bad news on TV and read a good book? Make time to make simple changes for your own health and the health of your family.

6- Know your farmer. Say “No” to factory farming. For high quality health, we need high quality nutrition. If you consume meat make sure it’s produced in the best possible way, without toxins and hormones. Food plays a role in skin health and in skin cancer. In fact, a high meat intake significantly increases the risk of squamous cell carcinoma.(4)

7- Take care of  your Mitochondria. Mitochondria convert food molecules (like glucose) into a chemical called ATP. There is energy damage causing premature aging when mitochondria is disrupted. Ditch the trans-fats and energy drinks and drink more pure clean water. Your brain and skin thrive on water.

8- Your skin needs sunshine, not a sunburn. Sunshine can rejuvenate, but, using toxic suntan oils and laying out in the sun all day can harm.

9- Get a grip on cravings and balance your adrenals. As the destructive nature of adrenal hormones overrides the body, we lose our connective tissue.(causing us to look older) This can lead to diabetes in the skin. (Altered skin wound healing.)(5)

10- Having a healthy gut means more than simply being free of annoyances like diarrhea or Gerd. It helps to consume fermented foods for healthy microbiome. A healthy microbiome is absolutely central to our immune function and our skin health.

11- No matter how old or young we are, we need our skin to go through the rejuvenating process when sleeping. Design your bedroom for optimal rest and relaxation.

12- Not only is our skin a barometer of what goes on inside your body, our skin hears everything we do. (It absorbs our everyday stressors and tension, while it protects our organs.) The crosstalk between epidermis, dermis as well as brain and endocrine organs are what should spark our interest when caring for our skin.(6) When we break the skins ability to protect us- it breaks down and can make us old before our time. This is premature aging!

There is beauty in everything that ages, but understand we don’t have to be sick as a side-effect of aging.

Connie Rogers is a Certified Integrative Nutritional Holistic Health Coach and Published Author, Certified Cosmetologist & Skin Health Expert, Gluten-Free Practitioner and Reiki Master. You can find her book on Amazon-Path to a Health Mind & Body    website-www.bitesizepieces.net

Prevent age stealers: https://bitesizepieces.leadpages.co/age-stealers-presentation/

footnotes:

1-https://www.ncbi.nlm.nih.gov/pubmed/15624317

2- https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3026597/

3- https://www.ncbi.nlm.nih.gov/pubmed/21547645

4- http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2836433/  see also https://www.ncbi.nlm.nih.gov/pubmed/17490979/

5- http://www.ncbi.nlm.nih.gov/pubmed/11423485

6- https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3051846/

Prevent Age Stealers… Use these ’12 Keys’ to increase quality of life and get your glow back

The small keys to immortality

Telomeres are simple in structural terms, but play a crucial role: they’re not genes, they don’t code for anything, but cells cannot reproduce without them. They are also key to ageing.

“Every time the cells go through a division and the chromosome is reproduced, it loses telomeres,” Rutherford explains. Our cells find it hard to copy the very tips of chromosomes and, slowly, the aglet is worn away from the end of the lace; the telomeres are depleted through repeated division.

“This seems to be a key part of why cells die during the ageing process,” Rutherford says. This eventual death of cells is called senescence, from the Latin senex, meaning old man. “When a cell gets to the point that it has no telomeres left, it won’t be able to divide again. Or if it does, it’ll be messed up.”

So the next question is, if we can restore the telomeres in elderly cells, might that stop them – and us – getting old?

That’s been the goal of researchers for a while. In the Nineties, Dr Elizabeth Blackburn and colleagues at the University of California won the Nobel prize for the discovery of telomerase, an enzyme that replenishes the telomeres in cells. The enzyme is usually “switched off”, but there is one kind of cell in which it demonstrates its potential in an often lethal way. “In cancer cells,” says Dr Kat Arney, a scientist at Cancer Research UK, “telomerase is switched on. And they become immortal.” Cancer cells can divide repeatedly without ever growing old, but this sort of uncontrolled growth has the unfortunate side-effect of killing the organism it’s growing in.

Telomerase may have a role to play in future treatments, according to Rutherford. “Telomeres are undoubtedly a key part of ageing, and the discovery of telomerase was a significant moment in understanding why cells don’t continue to reproduce forever, why they’re not immortal. If we could target that process of losing telomeres, with drugs or whatever, you could possibly maintain a longer shelf-life for a cell, it could continue reproducing far longer than its natural life. I don’t think it’s just science fiction; I think it’s genuinely a worthy topic of research with therapeutics in mind.”

In the shorter term, says Arney, telomere research may play a part in the fight against cancer. Since telomerase is found in cancer cells, some scientists thought they could target cancer cells in this way. “There was a massive trial into a vaccine against telomerase, called TeloVac, training the immune system to recognise it so that it would detect it in cancer cells,” says Arney. The trial showed no improvement over the standard chemotherapy treatment for the pancreatic cancer it targeted, but there is hope for other treatments in future.

Second, telomere loss is itself associated with cancers. When a cell’s telomeres are depleted, the chromosomes’ ends flap loose. “Cells hate loose ends of DNA,” says Arney. “If they come across some, they try to stitch them together, because they assume something’s gone wrong.” And if they stitch two flapping chromosomes together, “you get all sorts of mutations”, often causing cancer. Understanding telomerase could help prevent this.

As the function of telomeres becomes better understood, some private companies including Telomere Health and Life Length have begun offering to measure an individual’s chromosomal length, and so determine their “biological age”, as opposed to the chronological age. It is known that people with shorter telomeres are more likely to suffer various diseases, including diabetes, Parkinson’s and Alzheimer’s, and tend to die younger. One study found that people with shorter telomeres were eight times more likely to die from an infection and three times more likely to die from a heart attack.

The trouble is, says Rutherford, that the history of biology warns us that things are always complicated. “In biology, there is never one thing. For every rule that we have, we find a bunch of exceptions to it. And with genetics in particular, we’ve revealed a picture of complexity, rather than simplicity. In physics, things tend to get simpler the more we understand them. In biology, they tend to get more complex. Biology is messy.”

Telomeres “aren’t the only show in town”, says Rutherford, and there are many other research projects under way, from the serious to the seriously wacky. Research, again at the University of California, found that the lifespan of the roundworm C. elegans could be doubled by modifying two genes involved in cell repair, while this year the maverick scientist Craig Venter began a huge project sequencing human genes to find the ones that control longevity. And restricting the calorific intake of mice has been shown to extend their lives, sometimes dramatically. “Humans are trying that – there’s a weird cult in California of calorific restriction people, who eat some desperately low number of calories a day,” says Rutherford. There are two problems with this: one, individuals tend to be severely immuno‑compromised because of poor nutrition and get ill very easily, and two – “they all look absolutely miserable,” he says. “They eat about four pieces of lettuce a day. They might live forever, but they’re going to have an awful quality of life.”

There is a question, of course, over how far we would want to extend life. As Rutherford points out, merely extending old age would not be much of an achievement – the goal must be to extend the healthy years of life. And even if we can manage that, there would be ethical questions; life expectancy is already linked to class and wealth, but this would make that link direct and explicit. Of course, the world is already struggling with overpopulation and ageing societies.

That won’t stop scientists, though, and to date they agree that telomere research is the most promising avenue for extending life. “If we can slow the process of telomere loss, the fundamental cap on how often your cells can reproduce might be removed, or at least reduced,” Rutherford says.

It’s not an elixir of life; there will be no elixir of life. But this is the best science can do at providing one.

The small keys to immortality

The tiny keys to immortality

Telomeres are simple in structural terms, but play a crucial role: they’re not genes, they don’t code for anything, but cells cannot reproduce without them. They are also key to ageing.

“Every time the cells go through a division and the chromosome is reproduced, it loses telomeres,” Rutherford explains. Our cells find it hard to copy the very tips of chromosomes and, slowly, the aglet is worn away from the end of the lace; the telomeres are depleted through repeated division.

“This seems to be a key part of why cells die during the ageing process,” Rutherford says. This eventual death of cells is called senescence, from the Latin senex, meaning old man. “When a cell gets to the point that it has no telomeres left, it won’t be able to divide again. Or if it does, it’ll be messed up.”

So the next question is, if we can restore the telomeres in elderly cells, might that stop them – and us – getting old?

That’s been the goal of researchers for a while. In the Nineties, Dr Elizabeth Blackburn and colleagues at the University of California won the Nobel prize for the discovery of telomerase, an enzyme that replenishes the telomeres in cells. The enzyme is usually “switched off”, but there is one kind of cell in which it demonstrates its potential in an often lethal way. “In cancer cells,” says Dr Kat Arney, a scientist at Cancer Research UK, “telomerase is switched on. And they become immortal.” Cancer cells can divide repeatedly without ever growing old, but this sort of uncontrolled growth has the unfortunate side-effect of killing the organism it’s growing in.

Telomerase may have a role to play in future treatments, according to Rutherford. “Telomeres are undoubtedly a key part of ageing, and the discovery of telomerase was a significant moment in understanding why cells don’t continue to reproduce forever, why they’re not immortal. If we could target that process of losing telomeres, with drugs or whatever, you could possibly maintain a longer shelf-life for a cell, it could continue reproducing far longer than its natural life. I don’t think it’s just science fiction; I think it’s genuinely a worthy topic of research with therapeutics in mind.”

In the shorter term, says Arney, telomere research may play a part in the fight against cancer. Since telomerase is found in cancer cells, some scientists thought they could target cancer cells in this way. “There was a massive trial into a vaccine against telomerase, called TeloVac, training the immune system to recognise it so that it would detect it in cancer cells,” says Arney. The trial showed no improvement over the standard chemotherapy treatment for the pancreatic cancer it targeted, but there is hope for other treatments in future.

Second, telomere loss is itself associated with cancers. When a cell’s telomeres are depleted, the chromosomes’ ends flap loose. “Cells hate loose ends of DNA,” says Arney. “If they come across some, they try to stitch them together, because they assume something’s gone wrong.” And if they stitch two flapping chromosomes together, “you get all sorts of mutations”, often causing cancer. Understanding telomerase could help prevent this.

As the function of telomeres becomes better understood, some private companies including Telomere Health and Life Length have begun offering to measure an individual’s chromosomal length, and so determine their “biological age”, as opposed to the chronological age. It is known that people with shorter telomeres are more likely to suffer various diseases, including diabetes, Parkinson’s and Alzheimer’s, and tend to die younger. One study found that people with shorter telomeres were eight times more likely to die from an infection and three times more likely to die from a heart attack.

The trouble is, says Rutherford, that the history of biology warns us that things are always complicated. “In biology, there is never one thing. For every rule that we have, we find a bunch of exceptions to it. And with genetics in particular, we’ve revealed a picture of complexity, rather than simplicity. In physics, things tend to get simpler the more we understand them. In biology, they tend to get more complex. Biology is messy.”

Telomeres “aren’t the only show in town”, says Rutherford, and there are many other research projects under way, from the serious to the seriously wacky. Research, again at the University of California, found that the lifespan of the roundworm C. elegans could be doubled by modifying two genes involved in cell repair, while this year the maverick scientist Craig Venter began a huge project sequencing human genes to find the ones that control longevity. And restricting the calorific intake of mice has been shown to extend their lives, sometimes dramatically. “Humans are trying that – there’s a weird cult in California of calorific restriction people, who eat some desperately low number of calories a day,” says Rutherford. There are two problems with this: one, individuals tend to be severely immuno‑compromised because of poor nutrition and get ill very easily, and two – “they all look absolutely miserable,” he says. “They eat about four pieces of lettuce a day. They might live forever, but they’re going to have an awful quality of life.”

There is a question, of course, over how far we would want to extend life. As Rutherford points out, merely extending old age would not be much of an achievement – the goal must be to extend the healthy years of life. And even if we can manage that, there would be ethical questions; life expectancy is already linked to class and wealth, but this would make that link direct and explicit. Of course, the world is already struggling with overpopulation and ageing societies.

That won’t stop scientists, though, and to date they agree that telomere research is the most promising avenue for extending life. “If we can slow the process of telomere loss, the fundamental cap on how often your cells can reproduce might be removed, or at least reduced,” Rutherford says.

It’s not an elixir of life; there will be no elixir of life. But this is the best science can do at providing one.

The tiny keys to immortality